Hypertension / diabetes complications – manageable!
High blood pressure (hypertension) is the most common, yet most underestimated, symptom of systemic heart and kidney disease worldwide, affecting approximately 1.5 billion people. In Germany, 20 to 30 million people are affected. Hypertension is caused by inflammation as a physical reaction to individual stress and/or environmental influences such as particulate matter, chemicals, and contaminated food chains. Persistent hypertension is a sign of severe disruption of the endothelium in the blood vessels and of fibrosis. According to current medical knowledge, both phenomena occur in parallel, including in the heart and kidneys. Causalities cannot be determined within this complex molecular system.
In the early stages of the disease, effective medical treatments to prevent serious consequences such as heart attacks, dialysis and premature loss of life are neither possible nor have the diseases been defined and detected early.Almost every diabetic also suffers from hypertension.
High blood pressure is the body's reaction to chronic inflammation and fibrosis. The immune system is overactivated. With increasing damage to the inner wall cells, the endothelium, the inner wall of arteries, for example, thickens, which in turn increases the pressure of the blood flow. At the same time, collagens proliferate outside the blood vessels. These collagens have a stabilizing function and now themselves increase the external pressure on the blood vessels.
The kidneys play a central role in regulating blood pressure; they filter the blood. If chronic inflammation and/or fibrosis are not successfully treated, endothelial damage spreads throughout the blood vessels. Fibrosis also spreads, and normal tissue is replaced by scar tissue. Kidney function continuously declines until this process can no longer be stopped. This is the current diagnostic and therapeutic dilemma.
Increased blood pressure damages the overworked heart, causing the heart muscles to thicken. The blood vessels in and around the heart constrict, leading to a slowing of blood flow, a reduced supply (of oxygen, energy carriers, etc.), and further increased strain on the heart. In turn, damage to the endothelium and fibrosis continuously damage the organ. Without systemic and early analysis of this cellular, protein-controlled system that controls hypertension and the cardiovascular-renal system, efficient and successful treatment is impossible.